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Supplementary Material for: RAGE Does Not Contribute to Renal Injury and Damage upon Ischemia/Reperfusion-Induced Injury

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posted on 04.11.2011, 00:00 by Dessing M.C., Pulskens W.P., Teske G.J., Butter L.M., van der Poll T., Yang H., Tracey K.J., Nawroth P.P., Bierhaus A., Florquin S.
The receptor for advanced glycation end products (RAGE) mediates a variety of inflammatory responses in renal diseases, but its role in renal ischemia/reperfusion (I/R) injury is unknown. We showed that during renal I/R, RAGE ligands HMGB1 and S100B are expressed. However, RAGE deficiency does not affect renal injury and function upon I/R-induced injury.


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